Asia Joe Posted February 20 Posted February 20 September 4, 2017 Study: Coconut Oil Reverses Non-Alcoholic Fatty Liver Disease Total Views : 1,632 by Paul Fassa Health Impact News Non-alcoholic fatty liver disease (NAFLD) is defined medically as macrovesicular steatosis, or abnormal retention of lipids (fats) sufficient and large enough to distort or replace the nuclei of liver cells among those who consume less than 20 grams (.7 ounces) of alcohol per day. NAFLD, unknown prior to 1980, has become our largest liver health issue nationally and throughout most industrialized nations. Fatty liver disease affects metabolism and usually manifests as obesity and insulin resistance, which are direct co-factors for type 2 diabetes. It can also progress to inflammation of the liver, or non-alcoholic steatohepatitis (NASH), leading to cirrhosis. Ironically, a saturated fat (virgin coconut oil) may prove to be the safest and most effective treatment for NAFLD according to a study published recently (September 2017) in the Journal of the Science and Agriculture of Food. The study, “Virgin coconut oil reverses hepatic steatosis by restoring redox homeostasis and lipid metabolism in male Wistar rats,” noted in its background statement that more nutraceutical offerings are being sought for reversing fatty liver disease. Recent Study Proving Virgin Coconut Oil Efficacy and Safety for Reversing NAFLD The researchers noted that earlier studies had demonstrated virgin coconut oil could help prevent NAFLD. This new study intended to see if virgin coconut oil could also reverse existing non-alcoholic fatty liver disease. (Pharmaceutical medications, with side effects, are not working to stem the tide of rising NAFLD.) After four weeks on a high fructose diet, the rats developed non-alcoholic fatty liver disease, or hepatosteotosis. This was confirmed by observing the rats’ hyperglycemia (high blood sugar), reduced glucose tolerance (medical term for a zone between high blood sugar and diabetes), dyslipidemia (high blood lipids – triglycerides, etc.,) and hepatic macrovesicles (lipid displacements of liver cell nuclei). Although the rats could possibly eventually recover from hepatosteatosis (NAFLD) if taken off the high fructose diet, their recovery was accomplished more rapidly by feeding them virgin coconut oil (VCO). Their HDL (high density lipoprotein) or “good” cholesterol blood levels quickly increased by over 50 percent. Liver triglycerides were reduced 78 percent and blood serum triglycerides were reduced almost 52 percent. The liver is an important source of glutathione (GHS), a vitally important antioxidant that can be replenished and recycled only by a healthy liver. The rats glutathione levels also increased, resulting in increased antioxidant enzyme activities. Amazingly, liver damage was also repaired by administering virgin coconut oil. Lowered liver enzyme readings were also observed after feeding the rats VCO, confirming that their livers were returning to normal function. The researchers concluded: The study indicates that VCO [virgin coconut oil] can be used as a nutraceutical against hepatosteatosis [non-alcoholic fatty liver disease]. (Study abstract) The virgin coconut oil amounts used in this study were not made available in the abstract, however, earlier studies for using virgin coconut oil to reverse candida overgrowth and help reverse dementia indicate an estimated 3-3 1/2 tablespoons daily should work therapeutically for humans. (Source) The liver easily metabolizes coconut oil’s medium chain triglycerides or fatty acids (MCTs) and converts them into ketones which are easily metabolized for energy by the brain when glucose metabolism has faded, evidenced by use with dementia and other neurological issues. What about High Fructose Corn Syrup Effects on the Liver? The high fructose corn syrup diet was emphasized earlier in this article for a reason, to point out that HFCS was used to induce non-alcoholic fatty liver disease in the rats. High fructose corn syrup (HFCS) was developed as a sweetener to replace refined sugar as far back as the 1950s. Its use was not widespread until the 1970s. Around 1984, HFCS became the go-to sweetener in Pepsi and Coke as well as other beverages and processed food items, including pastries, candies, and even several unsweetened processed junk foods. According to an earlier study attempting to provide insights into treating non-alcoholic fatty liver disease, NAFLD was largely unknown prior to 1980 but is now recognized as the most common chronic liver disease in the U.S. and many other parts of the world. (Source) Is the correlation evident yet? Dr. Robert Lustig has certainly made this correlation obvious with his viral YouTube video lecture, “Sugar, the Bitter Truth,” where he explains how the rise in added refined sugar and fructose is the true culprit responsible for obesity, diabetes, and heart disease (not saturated fats, the usual suspect held by mainstream medicine and nutrition). HFCS is synthetically derived from processing corn sugar. The process creates a very concentrated form of fructose that is accompanied with some sucrose. This makes the corn sugar even sweeter and less expensive than cane sugar. Many argue that HFCS is the same as sugar but that is incorrect. The liver cannot metabolize highly concentrated synthetically derived fructose, it becomes fat that is mostly stored in the liver. This is the crux of NAFLD. The timelines of HFCS use and increasing NAFLD cases are related. HFCS can be disguised in USA processed food and beverage products as: Maize syrup Glucose syrup Glucose/Fructose syrup Tapioca syrup Dahlia syrup Fruit fructose Crystalline fructose Corn syrup In Canada, they call it Glucose-Fructose. In Europe, they call it Isoglucose or Glucose-Fructose Syrup or Fructose-Glucose Syrup. The process to create HFCS transmits some mercury into the final product. Another side effect from HFCS is the damage to leptin sensitivity. This hormone lets you know when you’ve had enough to eat and also contributes toward normal metabolism. (Source) If you have experienced some HFCS liver damage from past or current dietary indulgences, ignorance, or label reading negligence, you can educate yourself now and take advantage of virgin coconut oil to get rid of any trace of non-alcoholic fatty liver disease that is manifesting as poor digestion, sluggish metabolism, and obesity, even if it has already become prediabetes or type 2 diabetes. We are fortunate to know about the merits of virgin coconut oil that this site promotes. You may access many other informative articles on the health benefits of coconut oil here. Quote
Asia Joe Posted February 20 Author Posted February 20 https://pubmed.ncbi.nlm.nih.gov/28862329/ At the bottom: Similar articles Virgin coconut oil maintains redox status and improves glycemic conditions in high fructose fed rats. Narayanankutty A, Mukesh RK, Ayoob SK, Ramavarma SK, Suseela IM, Manalil JJ, Kuzhivelil BT, Raghavamenon AC.J Food Sci Technol. 2016 Jan;53(1):895-901. doi: 10.1007/s13197-015-2040-8. Epub 2015 Sep 22.PMID: 26788013Free PMC article. Thermally Oxidized Coconut Oil as Fat Source in High-Fat Diet Induces Hepatic Fibrosis in Diabetic Rat Model. Gopinath V, Shamsitha MKA, Penarveettil Nair V, Seena P, Uppu RM, C Raghavamenon A.Cell Biochem Biophys. 2021 Sep;79(3):629-639. doi: 10.1007/s12013-021-01009-5. Epub 2021 Jun 9.PMID: 34106414 Virgin coconut oil supplementation attenuates acute chemotherapy hepatotoxicity induced by anticancer drug methotrexate via inhibition of oxidative stress in rats. Famurewa AC, Ufebe OG, Egedigwe CA, Nwankwo OE, Obaje GS.Biomed Pharmacother. 2017 Mar;87:437-442. doi: 10.1016/j.biopha.2016.12.123. Epub 2017 Jan 6.PMID: 28068634 Hepatic lipid accumulation: cause and consequence of dysregulated glucoregulatory hormones. Geisler CE, Renquist BJ.J Endocrinol. 2017 Jul;234(1):R1-R21. doi: 10.1530/JOE-16-0513. Epub 2017 Apr 20.PMID: 28428362Review. Dietary sphingolipids: potential for management of dyslipidemia and nonalcoholic fatty liver disease. Norris GH, Blesso CN.Nutr Rev. 2017 Apr 1;75(4):274-285. doi: 10.1093/nutrit/nux004.PMID: 28383715Review. See all similar articles Cited by Lauric Acid versus Palmitic Acid: Effects on Adipose Tissue Inflammation, Insulin Resistance, and Non-Alcoholic Fatty Liver Disease in Obesity. Saraswathi V, Kumar N, Gopal T, Bhatt S, Ai W, Ma C, Talmon GA, Desouza C.Biology (Basel). 2020 Oct 22;9(11):346. doi: 10.3390/biology9110346.PMID: 33105887Free PMC article. Dietary intake of saturated fatty acids and the risk of incident diabetes: associations by isocaloric substitutions in a nationwide Chinese cohort. Ye H, Wu F, Zhuang P, Liu X, Li Y, Zhang Y, Jiao J.Eur J Nutr. 2025 May 20;64(4):181. doi: 10.1007/s00394-025-03703-z.PMID: 40392293 A high-fat diet supplemented with medium-chain triglycerides ameliorates hepatic steatosis by reducing ceramide and diacylglycerol accumulation in mice. Mourad S, Abdualkader AM, Li X, Jani S, Ceddia RB, Al Batran R.Exp Physiol. 2024 Mar;109(3):350-364. doi: 10.1113/EP091545. Epub 2024 Jan 9.PMID: 38192209Free PMC article. In vivo antioxidant and hypoglycaemic potentials of Rivina humilis extract against streptozotocin induced diabetes and its complications in wistar rats. C R, Ghosh K, A SB, Rawal P, Pramanik S.J Diabetes Metab Disord. 2023 Aug 17;22(2):1373-1383. doi: 10.1007/s40200-023-01258-6. eCollection 2023 Dec.PMID: 37975104Free PMC article. Nutrients, Physical Activity, and Mitochondrial Dysfunction in the Setting of Metabolic Syndrome. Lemos GO, Torrinhas RS, Waitzberg DL.Nutrients. 2023 Feb 28;15(5):1217. doi: 10.3390/nu15051217.PMID: 36904216Free PMC article.Review. See all "Cited by" articles Quote
Asia Joe Posted February 20 Author Posted February 20 There is NO Medicine to repair a fatty liver. This is exciting ! EVCO is easy to take. Quote Amazingly, liver damage was also repaired by administering virgin coconut oil. Lowered liver enzyme readings were also observed after feeding the rats VCO, confirming that their livers were returning to normal function. You would think, in a land that has coconut trees everywhere, that this would be easy for me to buy in every grocery store. No Philippines is a major exporter of Coconut products. They would rather sell it to the export market at the higher profit, than the locals. It is available, just not as convent as apples in Washington state. Quote
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